Faculty Profiles

Rivers, C., Farber, C., Heath, M., Gonzales, E., Barrett, D. W., Gonzalez-Lima, F., & Lane, M. A. (2024). Dietary omega-3 polyunsaturated fatty acids reduce cytochrome c oxidase in brain white matter and sensorimotor regions while increasing functional interactions between neural systems related to escape behavior in postpartum rats. Frontiers in Systems Neuroscience, 18. https://doi.org/10.3389/fnsys.2024.1423966

Farber, C. M., Renteria, K., Ritter, J., Muraida, J., Rivers, C., McKenzie, A. L., … Lane, M. A. (2023). Comparison of maternal versus postweaning ingestion of a high fat, high sucrose diet on depression-related behavior, novelty reactivity, and corticosterone levels in young, adult rat offspring. Behavioural Brain Research, 455, 114677. https://doi.org/https://doi.org/10.1016/j.bbr.2023.114677

Teoh, C. M., Cooper, A., Lane, M. A., Zhu, J., & Koh, G. Y. (2023). Maternal methyl donor nutrients supplementation to a high-fat high-sucrose diet modulates systemic vitamin D signaling and renal inflammation among adult offspring. Current Developments in Nutrition, 7(Supp 1), 752.

Cooper, A., Teoh, C. M., Renteria, K., Lane, M. A., Zhu, J., & Koh, G. Y. (2023). Colonic content from offspring born to dams of high-fat high-sucrose diet modulates IL-8 secretion in Lipopolysaccharide-stimulated HT-29 cells.

Teoh, C. M., Cooper, A., Renteria, K., Lane, M., Zhu, J., & Koh, G. Y. (2022). Supplementation of methyl-donor nutrients to a high-fat, high-sucrose diet during pregnancy and lactation normalizes circulating 25-dihydroxycholecalciferol levels and alleviates inflammation in offspring. Metabolites, 12(12), 1252. https://doi.org/10.3390/metabo12121252

Mathieu, V., Laguera, B., Masi, M., Dulanto, S. A., Bingham, T., Hernandez, L., … Lane, M. A. (2022). Amaryllidaceae Alkaloids Decrease the Proliferation, Invasion, and Secretion of Clinically Relevant Cytokines by Cultured Human Colon Cancer Cells. Biomolecules, 12.

Mathieu, V., Laguera, B., Masi, M., Dulanto, S. A., Bingham, T. W., Hernandez, L. W., … Lane, M. A. (2022). Amaryllidaceae Alkaloids Decrease the Proliferation, Invasion, and Secretion of Clinically Relevant Cytokines by Cultured Human Colon Cancer Cells. Biomolecules, 12(9), 1267. https://doi.org/10.3390/biom12091267

Teoh, C. M., Renteria, K., Cooper, A., Zhu, J., Lane, M., & Koh, G. Y. (2022). Supplementation of a high-fat high-sucrose diet with methyl-donor nutrients during pregnancy and lactation modulates the colonic vitamin D signaling pathway in weaned rats. Current Developments in Nutrition, 6(1), 1195. https://doi.org/https://doi.org/10.1093/cdn/nzac074.024

Pfister, E., Smith, R. J., & Lane, M. A. (2021). N-3 Polyunsaturated fatty acid ethyl esters decrease the invasion, but not the proliferation, of human colorectal cancer cells via a PI3K-dependent mechanism in vitro. Prostaglandins, Leukotrienes & Essential Fatty Acids, 167. https://doi.org/doi: 10.1016/j.plefa.2021.102273

Smith, R. J., Alvarez, C. G., Crixell, S. L., & Lane, M. A. (2021). The Food, Feelings, and Family Study: comparison of the efficacy of traditional methods, social media, and broadcast email to recruit pregnant women to an observational, longitudinal nutrition study. BMC Pregnancy and Childbirth, 21(203). https://doi.org/https://doi.org/10.1186/s12884-021-03680-1

Smith, R., Alvarez, C., Crixell, S., & Lane, M. A. (2021). The Food, Feelings, and Family Study: Comparison of the efficacy of traditional methods, social media, and broadcast email to recruit pregnant women to an observational, longitudinal nutrition study.

Smith, R. J., Pfister, E., & Lane, M. A. (2020). Ethyl ester n-3 polyunsaturated fatty acids decrease colorectal cancer cell invasion and proliferation via different mechanisms.

Pfister, E., & Lane, M. A. (2019). Abstract 1606: The ethyl ester forms of the n-3 fatty acids eicosapentaenoic acid and docosahexaenoic acid decrease the proliferation, invasion, and matrixmetalloproteinase activity of colorectal cancer cells. In Prevention, Early Detection, and Interception (pp. 1606–1606). American Association for Cancer Research. https://doi.org/10.1158/1538-7445.sabcs18-1606

Pfister, E., & Lane, M. A. (2019). The ethyl ester forms of the n-3 fatty acids eicosapentaenoic acid and docosahexaenoic acid decrease the proliferation, invasion, and matrixmetalloproteinase activity of colorectal cancer cells.

Jackson, C., Barrett, D. W., Gonzales, E., Shumake, J., Gonzalez-Lima, F., & Lane, M. A. (2018). Maternal omega-3 fatty acid intake during neurodevelopment does not affect pup behavior related to depression, novelty, or learning. Biomed Central Research Notes, 11(1), 812.

Pfister, E., & Lane, M. (2017). Free, ethyl- and methyl- ester eicosapentaenoic acid inhibit the proliferation of human and murine colon cancer cells in vitro. FASEB J, 71(Supple 1).

Martinez, M., Hurst, A., Reynolds, C. P., & Lane, M. (2017). The proliferation and invasion of low passage number human colon cancer cell lines is inhibited by retinoids. FASEB J, 71(Supple 1).

Jackson, C. S., Alhado, M., Gonzales, E., Shumake, J., Barrett, D., Gonzalez-Lima, F., & Lane, M. A. (2016). Maternal consumption of a diet lacking omega-3 fatty acids during development alters pup behavior and brain metabolism later in life. FASEB Journal.

Martinez, M. M., Hurst, A., Reynolds, C. P., & Lane, M. A. (2016). The proliferation and invasion of low passage number human colon cancer cell lines is inhibited by retinoids. FASEB J.

Rudzinski, W. E., Palacios, A., Ahmed, A., Lane, M. A., & Aminabhavi, T. M. (2016). Targeted delivery of small interfering RNA to colon cancer cells using chitosan and PEGylated chitosan nanoparticles. Carbohydrate Polymers, 147, 323–332. https://doi.org/10.1016/j.carbpol.2016.04.041

Gonzales, E., Barrett, D. W., Shumake, J., Gonzalez-Lima, F., & Lane, M. A. (2015). Omega-3 fatty acids improve behavioral coping to stress in multiparous rats. Behavioural Brain Research, 279, 129–138. https://doi.org/10.1016/j.bbr.2014.11.010

Applegate, C. C., & Lane, M. A. (2015). Role of retinoids in the prevention and treatment of colorectal cancer. World Journal of Gastrointestinal Oncology, 7(10), 184. https://doi.org/10.4251/wjgo.v7.i10.184

Jackson, C. S., Barrett, D. W., Shumake, J., Gonzalez-Lima, F., & Lane, M. A. (2014). Fatty Acid Consumption Decreases Offspring Reactivity to the Stress of a Novel Environment. In FASEB Journal.

Jackson, C. S., Barrett, D. W., Shumake, J., Gonzalez-Lima, F., & Lane, M. A. (2014). Maternal Omega-3 Fatty Acid Consumption Decreases Offspring Reactivity to the Stress of a Novel Environment. FASEB Journal.

Griffin Lengyel, J. N., Park, E. Y., Brunson, A. R., Pinali, D., & Lane, M. A. (2014). Phosphatidylinositol 3-Kinase Mediates the Ability of Retinol to Decrease Colorectal Cancer Cell Invasion. Nutrition and Cancer, 66(8), 1352–1361. https://doi.org/10.1080/01635581.2014.956258

Shumake, J., Barrett, D. W., Lane, M. A., & Wittke, A. J. (2014). Behavioral effects of bovine lactoferrin administration during postnatal development of rats. BioMetals, 27(5), 1039–1055. https://doi.org/10.1007/s10534-014-9735-6

Shumake, J., Barrett, D., Lane, M. A., & Wittke, A. (2013). Behavioral effects of bovine lactoferrin (bLF) supplementation in developing rats.

Gonzales, E. M., Barrett, D. W., Gonzalez-Lima, F., & Lane, M. A. (2013). Omega-3 fatty acid supplementation improves adaptive coping in a rat model of post-partum depression. In Faseb J.

Shumake, J., Barrett, D., Lane, M. A., & Wittke, A. (2013). Behavioral effects of bovine lactoferrin (bLF) supplementation in developing rats.

Gonzales, E. M., Barrett, D. W., Gonzalez-Lima, F., & Lane, M. A. (2013). Omega-3 fatty acid supplementation improves adaptive coping in a rat model of post-partum depression. FASEB Journal.

Park, E. Y., Pinali, D., Lindley, K., & Lane, M. A. (2012). Hepatic vitamin A preloading reduces colorectal cancer metastatic multiplicity in a mouse xenograft model. Nutrition and Cancer, 64, 732–740.

Olds, K., & Lane, M. A. (2012). Retinol tends to reduce activator protein-1 and nuclear factor kappa B mediated gene transcription in cultured human colorectal cancer cells. In Faseb J.

Olds, K., & Lane, M. A. (2012). Retinol tends to reduce activator protein-1 and nuclear factor kappa B mediated gene transcription in cultured human colorectal cancer cells. FASEB Journal.

Escamilla, J., Lane, M. A., & Maitin, V. (2012). Cell-free supernatants from probiotic Lactobacillus casei and Lactobacillus rhamnosus GG decrease colon cancer cell invasion in vitro. Nutrition and Cancer, 64(6), 871–878. https://doi.org/10.1080/01635581.2012.700758

Park, E. Y., Pinali, D., Lindley, K., & Lane, M. A. (2012). Hepatic Vitamin A Preloading Reduces Colorectal Cancer Metastatic Multiplicity in a Mouse Xenograft Model. Nutrition and Cancer, 64(5), 732–740. https://doi.org/10.1080/01635581.2012.687425

Drew, C. J., O’Reilly, K. C., Lane, M. A., & Bailey, S. J. (2011). Chronic administration of 13-cis- retinoic acid does not alter the number of serotoninergic neurons in the mouse raphe nuclei. Neuroscience, 172, 66–73.

Griffin, J. N., & Lane, M. A. (2011). Phosphatidylinositol 3-kinase mediates the ability of dietary vitamin A to decrease the incidence of hepatic colorectal cancer metastases in a mouse xenograft model.

Griffin, J. N., & Lane, M. A. (2011). Phosphatidylinositol 3-kinase mediates the ability of dietary  vitamin A to decrease the incidence of hepatic colorectal cancer metastases in a mouse xenograft model.

Griffin, J., Pinali, D., Olds, K., Lu, N., Appleby, L., Doan, L., & Lane, M. A. (2010). 13-Cis- retinoic acid decreases hypothalamic cell number in vitro. Neuroscience Research, 683, 185–190.

Voruganti, V. S., Cai, G., Klohe, D. M., Jordan, K. C., & Lane, M. A. (2010). Short-term weight loss in overweight/obese low-income women improves plasma zinc and metabolic syndrome risk factors. Journal of Trace Elements in Medicine and Biology, 24, 271–276.

Griffin, J. N., & Lane, M. A. (2010). Phosphatidylinositol 3-kinase mediates the ability of retinol to inhibit colon cancer cell invasion in vitro. In Faseb J.

Lane, M. A., Riha, P., & Shumake, J. (2010). 2010 Dietary vitamin A supplementation alters brain metabolism and network interactions in mice. In Faseb J.

Escamilla, J., Lane, M. A., & Maitin, V. (2010). Probiotic Lactobacilli decrease invasion of metastatic human colon cancer cells in vitro. In FASEB Journal.

Griffin, J. N., & Lane, M. A. (2010). Phosphatidylinositol 3-kinase mediates the Ability of retinol to inhibit colon cancer cell invasion in vitro. FASEB Journal.

Lane, M. A., Riha, P., & Shumake, J. (2010). 2010 Dietary vitamin A supplementation alters brain metabolism and network interactions in Mice. FASEB Journal.

Escamilla, J., Lane, M. A., & Maitin, V. (2010). Probiotic Lactobacilli decrease invasion of metastatic human colon cancer cells in vitro. FASEB Journal.

Voruganti, V. S., Cai, G., Klohe, D. M., Jordan, K. C., Lane, M. A., & Freeland-Graves, J. H. (2010). Short-term weight loss in overweight/obese low-income women improves plasma zinc and metabolic syndrome risk factors. Journal of Trace Elements in Medicine and Biology, 24(4), 271–276. https://doi.org/10.1016/j.jtemb.2010.05.001

Escamilla, J., Lane, M. A., & Maitin, V. (2010). Probiotic Lactobacilli Decrease Invasion of Metastatic Human Colon Cancer Cells In Vitro. The FASEB Journal, 24(S1). https://doi.org/10.1096/fasebj.24.1_supplement.928.21

Lane, M. A. (2009). Dietary vitamin A supplementation decreases colorectal cancer metastases in mice. In FASEB Journal (Vol. 23).

Escamilla, J., Lane, M. A., & Maitin, V. (2009). Probiotic Lactobacilli decrease invasion of metastatic human colon cancer cells in vitro.

Griffin, J. N., Park, E. Y., & Lane, M. A. (2009). Dietary vitamin A supplementation decreases colorectal cancer metastases in mice. FASEB Journal, 23.

Lane, M. A. (2009). Target: Colon Cancer. In Research Texas.

O’Reilly, K. C., Shumake, J., Bailey, S. J., Gonzalez-Lima, F., & Lane, M. A. (2009). Chronic 13-cis-retinoic acid administration disrupts network interactions between the raphe nuclei and the hippocampal system in young adult mice. European Journal of Pharmacology, 605(1–3), 68–77. https://doi.org/10.1016/j.ejphar.2008.12.037

Lane, M. A. (2008). Retinoic acid receptor-independent actions of the naturally occurring retinoids: Retinol, the retroretinoids, and 4-oxoretinol. In F. Columbus (Ed.), Vitamin A: New Research.

O’Reilly, K., Bailey, S. J., & Lane, M. A. (2008). Retinoid function in the adult CNS. Experimental Biology and Medicine, 233, 251–258.

Dillard, A., & Lane, M. A. (2008). Retinol increases beta-catenin/RXR alpha binding, leading to  the increased proteasomal degradation of beta-catenin and RXR alpha. Nutrition and Cancer, 60, 97–108.

Park, E. Y., Wilder, E. T., Chiptuk, J., & Lane, M. A. (2008). Retinol decreases phosphatidylinositol 3-kinase activity in colon cancer cells. Molecular Carcinogenesis, 47, 264–274.

Lane, M. A., Xu, J. E., Wilen, E. W., Sylvester, R., Derguini, F., & Gudas, L. J. (2008). LIF removal increases CRABPI and CRABPII transcripts in embryonic stem cells cultured in retinol or 4-oxoretinol. Molecular and Cellular Endocrinology, 280, 63–74.

O’Reilly, K., Bailey, S. J., & Lane, M. A. (2008). Retinoid-Mediated Regulation of Mood: Possible Cellular Mechanisms. Experimental Biology and Medicine, 233(3), 251–258. https://doi.org/10.3181/0706-mr-158

Lane, M. A., Xu, J., Wilen, E. W., Sylvester, R., Derguini, F., & Gudas, L. J. (2008). LIF removal increases CRABPI and CRABPII transcripts in embryonic stem cells cultured in retinol or 4-oxoretinol. Molecular and Cellular Endocrinology, 280(1–2), 63–74. https://doi.org/10.1016/j.mce.2007.09.010

Wheatley, K. E., Williams, E. A., Smith, N. C., Dillard, A., Park, E. Y., Nunez, N. P., … Lane, M. A. (2008). Low-Carbohydrate Diet Versus Caloric Restriction: Effects on Weight Loss, Hormones, and Colon Tumor Growth in Obese Mice. Nutrition and Cancer, 60(1), 61–68. https://doi.org/10.1080/01635580701510150

O’Reilly, K., Trent, S., Bailey, S. J., & Lane, M. A. (2007). 13-Cis-retinoic acid alters intracellular serotonin, increases 5-HT1A receptor, and serotonin reuptake transporter levels in vitro. Experimental Biology and Medicine, 232, 1195–1203.

Williams, E., Perkins, S., Smith, N., Hurstings, S., & Lane, M. A. (2007). Metabolic insights on carbohydrate vs. dietary energy restriction on weight loss in obese mice. Annals of Nutrition and Metabolism, 51, 232–243.

Park, E. Y., Wilder, E. T., & Lane, M. A. (2007). Retinol inhibits PI3K/Akt activity but does not  affect IRS-1 and PI3K levels in retinoic acid-resistant human colon cancer cells. In FASEB Journal (Vol. 21).

Dillard, A., & Lane, M. A. (2007). Retinol reduces beta-catenin protein in retinoic acid-resistant  colon cancer cells by increasing beta-catenin-RXR-alpha interaction in the nucleus and translocation into the cytosol. In FASEB Journal (Vol. 21).

O’Reilly, K. C., Bailey, S. J., & Lane, M. A. (2007). 13-Cis-retinoic acid alters serotonin reuptake transporter levels in RN46A-B14 raphe nuclei cells. In FASEB Journal (Vol. 21).

Park, E. Y., Wilder, E. T., & Lane, M. A. (2007). Retinol inhibits PI3K/Akt activity but does not affect IRS-1 and PI3K levels in retinoic acid-resistant human colon cancer cells. FASEB Journal, 21.

Dillard, A., & Lane, M. A. (2007). Retinol reduces beta-catenin protein in retinoic acid-resistant colon cancer cells by increasing beta-catenin-RXR-alpha interaction in the nucleus and translocation into the cytosol. FASEB Journal, 21.

O’Reilly, K. C., Bailey, S. J., & Lane, M. A. (2007). 13-Cis-retinoic acid alters serotonin reuptake transporter levels in RN46A-B14 raphe nuclei cells. FASEB Journal, 21.

Williams, E. A., Perkins, S. N., Smith, N. C. P., Hursting, S. D., & Lane, M. A. (2007). Carbohydrate versus Energy Restriction: Effects on Weight Loss, Body Composition and Metabolism. Annals of Nutrition and Metabolism, 51(3), 232–243. https://doi.org/10.1159/000104143

Dillard, A. C., & Lane, M. A. (2007). Retinol Increases β-Catenin-RXRα Binding Leading to the Increased Proteasomal Degradation of β-Catenin and RXRα. Nutrition and Cancer, 60(1), 97–108. https://doi.org/10.1080/01635580701586754

O’Reilly, K. C., Trent, S., Bailey, S. J., & Lane, M. A. (2007). 13-cis-Retinoic Acid Alters Intracellular Serotonin, Increases 5-HT_1AReceptor, and Serotonin Reuptake Transporter LevelsIn Vitro. Experimental Biology and Medicine, 232(9), 1195–1203. https://doi.org/10.3181/0703-rm-83

Park, E. Y., Wilder, E. T., & Lane, M. A. (2007). Retinol Inhibits the Invasion of Retinoic Acid–Resistant Colon Cancer Cells In Vitro and Decreases Matrix Metalloproteinase mRNA, Protein, and Activity Levels. Nutrition and Cancer, 57(1), 66–77. https://doi.org/10.1080/01635580701268238

Dillard, A. C., & Lane, M. A. (2007). Retinol decreases β‐catenin protein levels in retinoic acid‐resistant colon cancer cell lines. Molecular Carcinogenesis, 46(4), 315–329. https://doi.org/10.1002/mc.20280

O’Reilly, K. C., Shumake, J., Gonzalez-Lima, F., & Lane, M. A. (2006). Chronic administration of 13-cis-retinoic acid induces depression-related behavior in mice. Neuropsychopharmacology, 31, 1919–1927.

Dillard, A., & Lane, M. A. (2006). Retinol decreases beta-catenin levels in retinoic acid- resistant colon cancer cell lines via a RXR-mediated degradation pathway. In FASEB Journal (Vol. 20).

Bailey, S. J., O’Reilly, K. C., & Lane, M. A. (2006). 13-Cis-retinoic acid (Accutane) increases 5-HT1A receptor levels in vitro. In Journal of Psychopharmacology (5th ed., Vol. 20).

Park, E. Y., Wilder, E. T., & Lane, M. A. (2006). Retinol inhibits all-trans-retinoic acid-resistant colon cancer cell invasion by reducing matrixmetalloproteinase (MMP) levels and PI3K/Akt activity.

O’Reilly, K., Wilder, E., Bailey, S. J., & Lane, M. A. (2006). 13-Cis-retinoic acid alters serotonin and 5HT1A receptor levels and induces apoptosis in RN46A-B14 raphe nuclei cells.

Williams, E., Perkins, S., Smith, N., Hursting, S., & Lane, M. A. (2006). Carbohydrate restriction, dietary energy restriction and weight loss in obese mice: Metabolic insights. In FASEB Journal (Vol. 20).

Dillard, A., & Lane, M. A. (2006). Retinol decreases beta-catenin levels in retinoic acid- resistant colon cancer cell lines via a RXR-mediated degradation pathway. FASEB Journal, 20.

Bailey, S. J., O’Reilly, K. C., & Lane, M. A. (2006). 13-Cis-retinoic acid (Accutane) increases 5-HT1A receptor levels in vitro. Journal of Psychopharmacology, 20(5).

Park, E. Y., Wilder, E. T., & Lane, M. A. (2006). Retinol inhibits all-trans-retinoic acid-resistant colon cancer cell invasion by reducing matrixmetalloproteinase (MMP) levels and PI3K/Akt activity.

O’Reilly, K. C., Wilder, E., Bailey, S. J., & Lane, M. A. (2006). 13-Cis-retinoic acid alters serotonin and 5HT1A receptor levels and induces apoptosis in RN46A-B14 raphe nuclei cells.

Williams, E., Perkins, S., Smith, N., Hursting, S., & Lane, M. A. (2006). Carbohydrate restriction, dietary energy restriction and weight loss in obese mice: Metabolic insights. FASEB Journal, 20.

Bailey, S. J., & Lane, M. A. (2006). Retrieved from http://www.schizophreniaforum.org/pap/comsearch.asp?commentator=Lane,Michelle%20A

O’Reilly, K. C., Shumake, J., Gonzalez-Lima, F., Lane, M. A., & Bailey, S. J. (2006). Chronic Administration of 13-Cis-Retinoic Acid Increases Depression-Related Behavior in Mice. Neuropsychopharmacology, 31(9), 1919–1927. https://doi.org/10.1038/sj.npp.1300998

Lane, M. A. (2006). Acne Drug Roaccutane (Accutane) Linked to Depression. In Toronto Daily News.

Lane, M. A. (2006). Acne treatment could be trigger factor in suicide, say researchers. In New Zealand Herald.

Lane, M. A. (2006). Acne-treating drug leads to depressive behavior: research. In People’s Daily China.

Lane, M. A. (2006). Acne Drug Linked to Depression. In BBC News.

Lane, M. A. (2006). Research shows link between acne drug and depression. In The Guardian, UK.

Lane, M. A. (2006). Mice Study finds link between depression and acne treatment. In The Telegraph, UK.

Lane, M. A. (2006). Depression fears over acne drugs. In Daily Mail, UK.

Lane, M. A. (2006). Ability of retinol to inhibit retinoic acid-resistant colon cancer growth and metastasis.

Park, E. Y., Dillard, A., Williams, E., Wilder, E. T., Pepper, M. R., & Lane, M. A. (2005). Retinol inhibits the growth of retinoic acid-resistant and sensitive colon cancer cells through a retinoic acid receptor-independent mechanism. Cancer Research, 65, 9923–9933.

Lane, M. A., & Bailey, S. J. (2005). Role of retinoid signaling in the adult brain. Progress in Neurobiology, 75, 275–293.

O’Reilly, K. C., Johnson, E., Shumake, J., Gonzalez-Lima, F., & Lane, M. A. (2005). Effects of chronic 13-cis-retinoic acid (Accutane) administration on depression-related behavior in mice.

Dillard, A., & Lane, M. A. (2005). Retinol decreases beta-catenin levels in retinoic acid-resistant colon cancer cell lines through increased protein degradation (Vol. 46).

Wilder, E. T., & Lane, M. A. (2005). Retinol and retinoic acid inhibit the migration and invasion of retinoic-acid resistant colon cancer cells in vitro (Vol. 46).

O’Reilly, K. C., Johnson, E., Shumake, J., Gonzalez-Lima, F., Lane, M. A., & Bailey, S. J. (2005). Effects of chronic 13-cis-retinoic acid (Accutane) administration on depression-related behavior in mice.

Dillard, A., & Lane, M. A. (2005). Retinol decreases beta-catenin levels in retinoic acid-resistant colon cancer cell lines through increased protein degradation, 46.

Wilder, E. T., & Lane, M. A. (2005). Retinol and retinoic acid inhibit the migration and invasion of retinoic-acid resistant colon cancer cells in vitro, 46.

Park, E. Y., Dillard, A., Williams, E. A., Wilder, E. T., Pepper, M. R., & Lane, M. A. (2005). Retinol Inhibits the Growth of All-Trans-Retinoic Acid–Sensitive and All-Trans-Retinoic Acid–Resistant Colon Cancer Cells through a Retinoic Acid Receptor–Independent Mechanism. Cancer Research, 65(21), 9923–9933. https://doi.org/10.1158/0008-5472.can-05-1604

Lane, M. A., & Bailey, S. J. (2005). Role of retinoid signalling in the adult brain. Progress in Neurobiology, 75(4), 275–293. https://doi.org/10.1016/j.pneurobio.2005.03.002

Wilder, E. T., Pepper, M. R., & Lane, M. A. (2004). Retinoic acid and retinol reduce the migration of RA-resistant colon cancer cells through a basement membrane.

Dillard, A., & Lane, M. A. (2004). Retinol decreases beta-catenin levels in retinoic acid-resistant colon cancer cell lines.

Dillard, A., & Lane, M. A. (2004). Retinol decreases beta-catenin protein levels in retinoic acid-resistant colon cancer cells.

Wilder, E. T., Pepper, M. R., & Lane, M. A. (2004). Retinoic acid and retinol reduce the migration of RA-resistant colon cancer cells through a basement membrane.

Dillard, A., & Lane, M. A. (2004). Retinol decreases beta-catenin levels in retinoic acid-resistant colon cancer cell lines.

Dillard, A., & Lane, M. A. (2004). Retinol decreases beta-catenin protein levels in retinoic acid-resistant colon cancer cells.

Dillard, A., Leland, J., Wilder, E., & Lane, M. A. (2003). Retinol inhibits the growth of retinoic acid-resistant colon cancer cells via a retinoic acid independent mechanism. In FASEB Journal (Vol. 17).

Dillard, A., Leland, J., Wilder, E., & Lane, M. A. (2003). Retinol inhibits the growth of retinoic acid-resistant colon cancer cells via a retinoic acid independent mechanism. FASEB Journal, 17.

Chen, A. C., Yu, K., Lane, M. A., & Gudas, L. J. (2003). Homozygous deletion of the CRABPI gene in AB1 embryonic stem cells results in increased CRABPII gene expression and decreased intracellular retinoic acid concentration. Archives of Biochemistry and Biophysics, 411(2), 159–173. https://doi.org/10.1016/s0003-9861(02)00732-4

Lane, M. A., Baldwin, R. L., & Jesse, B. W. (2002). Developmental changes in ketogenic activity and messenger RNA expression during sheep rumen development. Journal of Animal Science, 80, 1538–1544.

Latimer, P., Wilder, E. T., & Lane, M. A. (2002). Retinol inhibits growth and induces apoptosis in retinoic acid resistant HCT-116 colon cancer cells.

Lane, M. A., Wilen, E., & Gudas, L. J. (2002). Correlation of 4-oxoretinol production with expression of CRABPI and CRABPII transcripts in embryonic stem cells. In FASEB Journal (Vol. 16).

Ng, S., Lane, M. A., & Gudas, L. J. (2002). Role of Cyp26 in stem cell differentiation (Vol. 43).

Latimer, P., Wilder, E. T., & Lane, M. A. (2002). Retinol inhibits growth and induces apoptosis in retinoic acid resistant HCT-116 colon cancer cells.

Lane, M. A., Wilen, E., & Gudas, L. J. (2002). Correlation of 4-oxoretinol production with expression of CRABPI and CRABPII transcripts in embryonic stem cells. FASEB Journal, 16.

Ng, S., Lane, M. A., & Gudas, L. J. (2002). Role of Cyp26 in stem cell differentiation, 43.

Lane, M. A., Baldwin, R. L., & Jesse, B. W. (2000). Sheep rumen metabolic development in response to different dietary treatments. Journal of Animal Science, 78, 1990–1996.

Lane, M. A., Chen, A., Roman, S., Derguini, F., & Gudas, L. J. (2000). 4-Oxoretinol production from retinol in embryonic stem cells in increased by the addition of exogenous retinoids.

Lane, M. A., Chen, A., Roman, S., Derguini, F., & Gudas, L. J. (2000). . 4-Oxoretinol production from retinol in embryonic stem cells in increased by the addition of exogenous retinoids.

Lane, M. A., Chen, A., Roman, S., & Gudas, L. J. (1999). Retinol metabolism is induced in embryonic stem cells by the removal of leukemia inhibitory factor. In FASEB Journal (Vol. 13).

Lane, M. A., Chen, A., Roman, S., & Gudas, L. J. (1999). Retinol metabolism is induced in embryonic stem cells by the removal of leukemia inhibitory factor. FASEB Journal, 13.

Lane, M. A., Chen, A. C., Roman, S. D., Derguini, F., & Gudas, L. J. (1999). Removal of LIF (leukemia inhibitory factor) results in increased vitamin A (retinol) metabolism to 4-oxoretinol in embryonic stem cells. Proceedings of the National Academy of Sciences, 96(23), 13524–13529. https://doi.org/10.1073/pnas.96.23.13524

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